{"entity": "publication", "iuid": "016523f5e90c4b6f8258e6b4750542c5", "timestamp": "2026-09-08T10:05:39.839Z", "links": {"self": {"href": "https://publications-affiliated.scilifelab.se/publication/016523f5e90c4b6f8258e6b4750542c5.json"}, "display": {"href": "https://publications-affiliated.scilifelab.se/publication/016523f5e90c4b6f8258e6b4750542c5"}}, "title": "14-3-3\u03b6 turns TGF-\u03b2 to the dark side.", "authors": [{"family": "ten Dijke", "given": "Peter", "initials": "P"}, {"family": "van Dam", "given": "Hans", "initials": "H"}], "type": "journal article", "published": "2015-02-09", "journal": {"title": "Cancer Cell", "issn": "1878-3686", "volume": "27", "issue": "2", "pages": "151-153", "issn-l": "1535-6108"}, "abstract": "TGF-\u03b2/SMAD signaling has long been known to exhibit a dual role in cancer, questioning what determines its context-dependent functions. In this issue of Cancer Cell, Xu and colleagues describe a critical role of the adaptor protein 14-3-3\u03b6 in modulating SMAD activities by changing its interaction partners during breast cancer progression.", "doi": "10.1016/j.ccell.2015.01.005", "pmid": "25670073", "labels": [], "xrefs": [{"db": "pii", "key": "S1535-6108(15)00020-3"}], "notes": [], "created": "2026-08-21T11:14:56.883Z", "modified": "2026-08-21T11:14:56.915Z"}